Ammendola S, Mario Di Loreto, Anna Scotto d'Abusco.Modulatory effects of a herbal composition on Saos-2 cells reveal its phlebotonic activity. 2017 J. Am. Coll. Nutrition in print

Ammendola, S., Stoppoloni, D, Di Loreto, M, and Scotto d’Abusco,A.. A Nutraceutical Composition Decreases CPK Levels in Saos-2 Cells and in Patients with Elevated Serum Levels of This Enzyme. 2016 J. Am. Coll. Nutrition. Aug;35(6):559-567

Cilia E, Teso S, Ammendola S, Lenaerts T., Passerini A.: Predicting virus mutations through statistical relational learning. BMC Bioinformatics 2014;15:309. doi: 10.1186/1471-2105-15-309- Predicting virus mutations through statistical relational learning.BMC Bioinformatics. 2014;15:309. doi: 10.1186/1471-2105-15-309.

Ammendola S., Giusti A.M., Masci A., Mosca L., Saso L. and Bovicelli P. Synthesized Hydroxytyrosol and its Acetate influece the amount of sulfhydryl and carbonyl groups in SH-SY5Y neuroblastoma exposed to hydrogen peroxide. Jour. Sci. Indust. Res., 2011; 70: 929-937.

Radogna F, Paternoster L, De Nicola M, Cerella C, Ammendola S, Bedini A, Tarzia G, Aquilano K, Ciriolo M, Ghibelli L. : Rapid and transient stimulation of intracellular reactive oxygen species by melatonin in normal and tumor leukocytes. Toxicol Appl Pharmacol. 2009; 239(1): 37-45.

Ammendola S., Mosca L., and Bovicelli P.: An expedient synthesis of 2,5-dihydroxytyrosol and studies on its effects on cell growth inhibition ARKIVOC 2008; 8: 105-115.

Bovicelli, P., Antonioletti, R., Mancini, S., Causio, S., Borioni, G., Ammendola, S. and Barontini, M. (2007) 'Expedient Synthesis of Hydroxytyrosol and its Esters', Synthetic Communications, 37(23): 4245 – 4252.

Sagratini G, Fabbri A, Marucci G, Ricciutelli M, Vittori S and Ammendola S: HPLC–MS validation of QualisaFoo^® biosensor kit for cost-effective control of acrylamide levels in Italian coffee- Food Control Oct; 2007; 18: 1267-1271.

Cerella C, Mearelli C, Ammendola S, De Nicola M, D'Alessio M, Magrini A, Bergamaschi A, Ghibelli L. Molecular determinants involved in the increase of damage-induced apoptosis and delay of secondary necrosis due to inhibition of mono(ADP-ribosyl)ation. Ann N Y Acad Sci. 2006 Dec;1090:50-8.